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COVID Fatigue 2.0 Triage With everything people are facing now, sometimes the best strategy is to just do the little things you need to do to survive, Hermanson said. When we feel like there is so much we can’t do, we have to shift our focus to what we can do. Some of her suggestions:... ~Take it day by day, or moment by moment: Don’t look too far down the road, she said. Realize you will have good days and bad days, or good moments and bad moments. Realize these things can come in waves. It’s OK to say, ‘Right now, it’s bad.’~Just hang in there and ask, ‘What can I do to help feel better, or less bad?’ ~Be compassionate with yourself: Don’t expect perfection and don’t wallow in mistakes or missed chances. Nobody prepared us for this, Hermanson said. There wasn’t a class in high school called How to Get Through a Pandemic. We’re all making this is up as we go along. ~Be creative about finding things to look forward to: It could be a walk (when the smoke clears), or finding repeats of a TV series you love, or, as in Hermanson’s case, gathering a group of friends for a Zoom trivia night. We write down our answers then show them. There’s a certain amount of honesty involved, she said. We have to remember the purpose is to have fun, not to win. ~Find reasons to laugh: There is a healthy physical reaction to laughing, Hermanson said. If nothing else works, put on your favorite comedy. ~Exercise: It’s still the No. 1 best thing we can do for coping, she said. It releases endorphins and gets some of the adrenaline out when the frustration builds up. Just go for a walk, if you can. If the smoke is bad, exercise indoors. Pull up a yoga or workout video. It helps so much. ~Look back, but carefully: Don’t think all the way back to last summer and those weeks you spent at the lake, Hermanson said. But think about the past few months. We’ve really come a good distance. If you had told me in March what we were about to go through, it would have felt overwhelming. But think about how far we’ve come. Look at all the things we’ve managed. Look at how resilient we’re becoming. https://health.ucdavis.edu//coping-with-covid-fat/2020/09

"The improved oxygenation in prone position that has been described in patients with COVID-19 could be explained mainly through vascular redistribution towards the areas of apparently healthy lung with a high V/Q ratio, rather than alveolar recruitment." https://ccforum.biomedcentral.com//10.1/s13054-020-03125-9

"Infection control specialists also often inquire about the relative contribution of airborne transmission compared to the other transmission modes (‘contact’ and ‘droplet’). Multiple studies provide strong evidence for indoor airborne transmission of viruses, particularly in crowded, poorly ventilated environments" https://www.sciencedirect.com//artic/pii/S0160412020317876

"When it comes to COVID-19, the evidence overwhelmingly supports aerosol transmission, and there are no strong arguments against it. For example, contact tracing has found that much COVID-19 transmission occurs in close proximity, but that many people who share the same home with an infected person do not get the disease. To understand why, it is useful to use cigarette or vaping smoke (which is also an aerosol) as an analog. Imagine sharing a home with a smoker: if you stood... close to the smoker while talking, you would inhale a great deal of smoke. Replace the smoke with virus-containing aerosols, which behave very similarly, and the impact is similar: the closer you are to someone releasing virus-carrying aerosols, the more likely you are to breathe in larger amounts of virus. We know from detailed, rigorous studies that when individuals talk in close proximity, aerosols dominate transmission and droplets are nearly negligible." https://time.com/5883081/covid-19-transmitted-aerosols/

"Through a lock-and-key-like interaction between an ACE2 receptor and a spike RBD, the virus gains entry into the cell, where it then transforms its new host into a coronavirus manufacturer. The researchers believed that if they could find nanobodies that impede spike-ACE2 interactions, they could prevent the virus from infecting cells." https://www.universityofcalifornia.edu//aeronabs-promise-p

"The U.S. just experienced its worst two-week stretch, with more newly confirmed cases than at any point since its coronavirus outbreak began in early 2020. From June 25 to July 8, 674,750 Americans were diagnosed with coronavirus, and the nation’s tally grew by one million cases over the span of the past month."

https://www.cidrap.umn.edu//few-spanish-us-studies-had-det

https://jamanetwork.com//jamaneurology_abdelmannan_2020_br Increasing reports of children developing systemic inflammatory response requiring intensive care (labeled pediatric multisystem inflammatory syndrome temporally associated with COVID-19) and a further group of children with a far less severe, Kawasaki-like disease, who respond to a variety of immunomodulatory treatments, suggest that despite the typically mild acute infection, children may be at high risk of a secondary inflammatory syndrome.

The phenotype of our cohort raises the possibility of a virus-specific immunological syndrome," the researchers concluded. "A plausible mechanism would be exposure of the immune system to new CNS [central nervous system] antigens as a result of blood-brain barrier damage from SARS-CoV-2, which causes endotheliopathy and leads to an immune-directed attack on the CNS." https://www.medpagetoday.com/infectiousdisea/covid19/87378

https://www.grapevine.org//Black-Racial-Justice-Giving-Cir

https://emergency.cdc.gov/c/calls/2020/callinfo_051920.asp